Zingiberis rhizoma: a comprehensive review on the ginger effect and efficacy profiles.
Phytomedicine.
2005 Sep;12(9):684-701.
PMID:16194058

Characterization of gingerol-related compounds in ginger rhizome (Zingiber officinale Rosc.) by high-performance liquid chromatography/electrospray ionization mass spectrometry.
Rapid Commun Mass Spectrom. 2005;19(20):2957-64.
PMID:16189817

Ghayur MN, Gilani AH.
Pharmacological basis for the medicinal use of ginger in gastrointestinal disorders.
Dig Dis Sci. 2005 Oct;50(10):1889-97.
PMID:16187193

16169024&query_hl=35">Jiang H, Xie Z, Koo HJ, McLaughlin SP, Timmermann BN, Gang DR.
Metabolic profiling and phylogenetic analysis of medicinal Zingiber species: Tools for authentication of ginger (Zingiber officinale Rosc.).
Phytochemistry. 2005 Sep 15; [Epub ahead of print]
PMID:16169024

Anderson FW, Johnson CT.
Complementary and alternative medicine in obstetrics.
Int J Gynaecol Obstet. 2005 Sep 15; [Epub ahead of print]
PMID:16168989

Sandhu DS, Heinrich M.
The use of health foods, spices and other botanicals in the Sikh community in London.
Phytother Res. 2005 Jul;19(7):633-42.
PMID:16161027

Shin SG, Kim JY, Chung HY, Jeong JC.
Zingerone as an antioxidant against peroxynitrite.
J Agric Food Chem. 2005 Sep 21;53(19):7617-22.
PMID:16159194

Ghayur MN, Gilani AH, Afridi MB, Houghton PJ.
Cardiovascular effects of ginger aqueous extract and its phenolic constituents are mediated through multiple pathways.
Vascul Pharmacol. 2005 Sep 9; [Epub ahead of print]
PMID:16157513

Marcus DM, Snodgrass WR.
Effectiveness and safety of ginger in the treatment of pregnancy-induced nausea and vomiting.
Obstet Gynecol. 2005 Sep;106(3):640; author reply 640-1. No abstract available.
PMID:16135602

Borrelli F, Capasso R, Izzo AA.
Effectiveness and safety of ginger in the treatment of pregnancy-induced nausea and vomiting.
Obstet Gynecol. 2005 Sep;106(3):640-1. No abstract available.
PMID:16135601

Boone SA, Shields KM.
Treating pregnancy-related nausea and vomiting with ginger.
Ann Pharmacother. 2005 Oct;39(10):1710-3. Epub 2005 Aug 30.
PMID:16131535

Gilani AH, Rahman AU.
Trends in ethnopharmocology.
J Ethnopharmacol. 2005 Aug 22;100(1-2):43-49.
PMID:16127805

Cady RK, Schreiber CP, Beach ME, Hart CC.
Gelstat Migraine (sublingually administered feverfew and ginger compound) for acute treatment of migraine when administered during the mild pain phase.
Med Sci Monit. 2005 Sep;11(9):PI65-9. Epub 2005 Aug 26.
PMID:16127373

Wang H, Ng TB.
An antifungal protein from ginger rhizomes.
Biochem Biophys Res Commun. 2005 Oct 14;336(1):100-4.
PMID:16125680

Shen CL, Hong KJ, Kim SW.
comparative effects of ginger root (Zingiber officinale Rosc.) on the production of inflammatory mediators in normal and osteoarthrotic sow chondrocytes.
J Med Food. 2005 Summer;8(2):149-53.
PMID:16117605

Grzanna R, Lindmark L, Frondoza CG.
Ginger--an herbal medicinal product with broad anti-inflammatory actions.
J Med Food. 2005 Summer;8(2):125-32.
PMID:16117603

Lim WC, Seo JM, Lee CI, Pyo HB, Lee BC.
Stimulative and sedative effects of essential oils upon inhalation in mice.
Arch Pharm Res. 2005 Jul;28(7):770-4.
PMID:16114490

Lopez P, Sanchez C, Batlle R, Nerin C.
Solid- and vapor-phase antimicrobial activities of six essential oils: susceptibility of selected foodborne bacterial and fungal strains.
J Agric Food Chem. 2005 Aug 24;53(17):6939-46.
PMID:16104824

Kim EC, Min JK, Kim TY, Lee SJ, Yang HO, Han S, Kim YM, Kwon YG.
[6]-Gingerol, a pungent ingredient of ginger, inhibits angiogenesis in vitro and in vivo.
Biochem Biophys Res Commun. 2005 Sep 23;335(2):300-8.
PMID:16081047

Weseler A, Geiss HK, Saller R, Reichling J.
A novel colorimetric broth microdilution method to determine the minimum inhibitory concentration (MIC) of antibiotics and essential oils against Helicobacter pylori.
Pharmazie. 2005 Jul;60(7):498-502.
PMID:16076074

Microbial origin of other chronic inflammatory conditions?

Oct 3, 2005 The Nobel Assembly at Karolinska Institutet Awards Prize

"Many diseases in humans such as Crohn's disease, ulcerative colitis, rheumatoid arthritis and atherosclerosis are due to chronic inflammation. The discovery that one of the most common diseases of mankind, peptic ulcer disease, has a microbial cause, has stimulated the search for microbes as possible causes of other chronic inflammatory conditions. Even though no definite answers are at hand, recent data clearly suggest that a dysfunction in the recognition of microbial products by the human immune system can result in disease development. The discovery of Helicobacter pylori has led to an increased understanding of the connection between chronic infection, inflammation and cancer." More

Inflammatory heart disease: A role for cytokines

Lupus. 2005;14(9):646-51.

Department of Pathology, Johns Hopkins Medical Institutions, Baltimore, MD, USA.

Inflammatory heart disease is a rising concern worldwide. Similar mechanisms link autoimmune diseases, including the association of increased disease with pro inflammatory cytokines and the importance of regulatory mechanisms in the control of chronic inflammation. Many pathogens including bacteria, protozoa and viruses have been associated with heart disease in patients, and are able to induce similar disease in animal models. Recognition of pathogens by the innate immune system leads to release of pro inflammatory cytokines that both reduce infection and increase chronic inflammatory heart disease. Elevated levels of pro inflammatory cytokines are able to overcome tolerance to chronic disease, indicating that environmental factors are important in determining progression to chronic heart disease. Understanding the mechanisms leading to chronic heart disease will be critical for developing effective therapies to reduce cardiac dysfunction and heart failure.

The relevance of selenium to immunity, cancer, and infectious / inflammatory diseases

Selenium is an essential trace element involved in several key metabolic activities via selenoproteins, enzymes that are essential to protect against oxidative damage and to regulate immune function. Selenium also may have other health benefits unrelated to its enzymatic functions. It may provide important health benefits to people whose oxidative stress loads are high, such as those with inflammatory or infectious diseases like rheumatoid arthritis or human immunodeficiency virus/acquired immunodeficiency syndrome, or who are at high risk for cancers, particularly prostate cancer. Some studies have generated compelling evidence that selenium is beneficial, either alone or in conjunction with other micronutrients. Additional data from large clinical trials that provide the highest level of evidence will be key to determining the benefits accrued at various selenium intake levels. When the strength of the evidence becomes sufficient, clinical health professionals will need to use dietary and clinical assessment methods to ensure that people at increased risk for cancer or inflammatory and infectious diseases can be appropriately advised about selenium intake. Source:Cancer Journal Diet Pract Res. 2005 Summer;66(2):98-102.

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